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An enzyme trafficking defect in two patients with primary hyperoxaluria type 1: peroxisomal alanine/glyoxylate aminotransferase rerouted to mitochondria
Moreover, overexpression of constitutively active STAT3 mutants (STAT3-S727D), but not wild-type STAT3, was sufficient to induce Vgf promoter activity (Fig
Int J Sci Res
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