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Both promote angiogenesis through different pathways (BPC-157 via VEGFR2-Akt-eNOS and TB-500 via VEGF/HIF-1a), potentially amplifying blood vessel formation beyond what either achieves alone

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However, when supplemental energy is needed, necessary energy demands may be provided by other metabolites, such as ketones, fatty acids, acetate, lactate, and certain amino acids [19]