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Cell-penetrating peptide TAT-HuR-HNS3 suppresses proinflammatory gene expression via competitively blocking interaction of HuR with its partners

10.1038/nmat3776 49 NiK.LanG.LinW

The new findings highlight the importance of glutathione mobility within the cell and the direct interaction of glutathione-associated enzymes with viral factors or vRNA essential for the full-fledged development or initiation of viral infection (particularly through interactions with UTR sites in vRNA)