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Hepatic steatosis in the setting of hepatitis C genotype 3 (HCV-3) is driven by viral influence on three major pathways: microsomal triglyceride transfer protein, sterol regulatory element-binding protein-1c, and peroxisome proliferator associated receptor-

Therefore, CUP-5 protein localizes to lysosomes at steady state in developing intestinal cells

To equilibrize the lipid motions and minimize flip-flops of the bilayer lipid molecules, we simulated the membrane for an additional 1 microsecond at a constant temperature and pressure of 300 K and 1 bar
