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It has been shown that TMAO causes an increase in electrical remodeling and inflammation of the ganglionated plexi, which speeds up the course of arrhythmias such as AF

Figure 4B further support that under the background of normal mice, the process of Pg -induced elevated plasma TMAO levels depends more on the oxidation of TMA in the liver rather than the generation of TMA in the intestine

As researchers continue to unravel these connections, the role of mitochondrial antioxidants and shuttles like L-carnitine becomes increasingly central to emerging treatment protocols, often paired with other respiratory and cellular supports

Preterm neonates develop carnitine deficiency because of impaired proximal renal tubule carnitine reabsorption and immature carnitine biosynthesis