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Its proposed mechanisms include the enhancement of lipid metabolism, improvement of insulin sensitivity, stabilization of hepatocyte function, and reduction of inflammation, fibrosis, and tumorigenesis

Cathepsin S regulates antitumor immunity through autophagic degradation of PD-L1 in colorectal cancer cells

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It has been demonstrated that NETs can induce pro-inflammatory and pro-angiogenic responses in human pulmonary artery endothelial cells (HPAEC) through MPO/H 2 O 2 -dependent activation of TLR4/NF-B signaling (161)