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Taken together, these studies indicate that aging is highly involved with the pathology of glaucoma

Available evidence suggests that ferroptosis integrates multiple upstream damage signals, including pathogenic bacterial infection, iron overload, and oxidative stress, and may exacerbate tissue destruction and inflammation amplification through positive feedback loops

GPER1 activation initiates multiple intracellular signaling cascades, such as the phosphoinositide 3-kinase (PI3K) pathway, mitogen-activated protein kinase (MAPK) pathways and the production of cyclic adenosine monophosphate (cAMP)

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