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Although early NAC treatment is beneficial in limiting the injury, continuous NAC treatment well beyond the injury phase can delay regeneration [72] mainly through impairment of mitochondrial biogenesis, an adaptive response whose role is to maintain or restore homeostasis following mitochondrial dysfunction [71]

Gastrointestinal symptoms often present years before motor symptoms in PD and evidence suggests gut microbes play a role in PD pathogenesis via the microbiota-gut-brain axis (52)

Pierozan P, Coln-Gonzlez AL, Biasibetti H, da Silva JC, Wyse A, Wajner M, Santamaria A (2018) Toxic synergism between quinolinic acid and glutaric acid in neuronal cells is mediated by oxidative stress: insights to a new toxic model