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This review will summarize what is currently known about the mechanisms underlying c-Myc regulation of cancer stemness and its involvement in drug resistance and offer an overview on the current progress and future prospects in therapeutically targeting c-Myc in both SCLC and NSCLC
Indeed, pretreated LPS/ATP-stimulated macrophages with DTT almost completely abolished GYY4137-induced inhibition of p65 transcriptional activity (Fig
doi: 10.1177/0003319709335028
