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In conclusion, our structural and functional analyses provide a large body of novel information for defining the molecular mechanism underlying the activation of c-MET in response to HGF and NK1 and paves the way for the eventual therapeutic intervention of diseases caused by aberrant activation of c-MET

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Electromyography and nerve conduction studies conducted 2 weeks after the accident confirmed a severe multiple trunk brachial plexopathy characterized by motor axon loss

For all outcomes, the study staff have received training before initiation of the project, and the staff will be used based on their skill level