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The pathological characteristics of ALI involve extensive inflammation resulting in harm to alveolar epithelial cells (AECs), breakdown of the pulmonary vascular endothelial barrier and subsequent accumulation of protein-rich inflammatory edema fluid within the alveolar spaces, ultimately causing diffuse alveolar damage (6)

Its excellent for prevention, cellular optimization, and enhancing surgical results, but severe structural changes may still require surgical intervention
Moreover, ATG and apoptosis induction had common starting condition with ATG underlying first cell response (survival) before commitment to apoptosis

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