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Description
BACH1 directly modulates numerous critical inflammatory and repairrelated genes in musclederived macrophages following cardiotoxin injury, including IGF1, SLC40A1, IL6, IL10, GDF3, PPARG, DUSP1, and CEBPB, potentially elucidating the delayed muscle regeneration phenotype observed in BACH1 KO mice
In addition, the number of G1-phase cells significantly increased after si-IGF-1R transfection, but the number of cells in the S and G2 phases noticeably decreased (Figures 6E,F
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