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Understanding how it actually works at a biological level, not just what it is claimed to do, is the foundation for making any responsible clinical decision about whether it belongs in a protocol
Spindle abnormalities were minimal at early time points but increased significantly by 24 hours in untreated oocytes
The pathophysiology of FGIDs is complex and involves changes in ENS structure and function, along with factors like inflammation and psychosocial stress[39]
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