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On the other hand, Dave and coworkers found a disruption of the Calu3 ALI monolayer infected with R001, N222.1, and N459.6 meningococcal strains, suggestive of a paracellular route of epithelial crossing (Dave et al., 2023)
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562894), BUV395CD45 (clone 30-F11
Anti-Inflammatory Reduces scalp micro-inflammation, which is a known trigger for telogen effluvium and thinning
