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The role of macrophages in acute and chronic wound healing and interventions to promote pro-wound healing phenotypes
Here we posit that E + Hg forms will disrupt the cell metabolism via blockage of cysteinyl residues involved in protein regulation via glutathionylation or nitrosylation
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At the same time, the bodys natural antioxidant enzymes, such as catalase and superoxide dismutase, are also damaged, which affects tyrosinase and sharply reduces its activity
