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Description
It is preset in mitochondrial respiratory chains and servs to control reactive oxygen species production via electron transport cycling processes such as oxidative phosphorylation
Several previous studies have suggested that the Mito K ATP channel activation promotes HIF-1 expression
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Importantly, Set7/9-dependent methylation of -catenin is enhanced under oxidative stress conditions, thus forming a regulatory mechanism for the WNT/-catenin signaling during the oxidative stress response 132
