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Ischemic postconditioning has been explored as a strategy to alleviate limb IR-induced renal injury, with studies revealing notable histopathological alterations in kidney tissues post injury [13]

Neutrophil apoptosis usually results from caspase activation through various pathways, and is characterized by changes in cell shape, pseudopod retraction, volume reduction, and chromatin condensation (102104), and observation of these specialized changes under a microscope indicate that apoptosis is occurring

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HBOT has garnered significant attention as a potential therapeutic intervention for arthritis, with support from several studies in the literature (207, 208)