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These results highlighted that SQH could be considered a promising therapeutic candidate for the treatment of DbCM
10.1016/j.ijsu.2014.07.015 79 TakhtfooladiM
NO modulates mitochondrial biogenesis through different routes, a selective one is by increasing the phosphorylated-active form of AMP-activated protein kinase (AMPK) that in turn induces PGC-1a signaling pathway [72]
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