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By triggering NLRP3 activation in human induced pluripotent stem cells (iPSC)-derived microglia through TLR2 engagement and mitochondrial damage, -synuclein species have been shown to directly contribute to innate immunological activation in PD [56, 57]
In the skin, these reactive aldehydes act as secondary messengers in redox-sensitive pathways, including NF-B and Nrf2, thus influencing gene expression, protein modifications, and enzymatic activity, which are essential for maintaining epidermal integrity and function
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