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Toward the estimation of the absolute quality of individual protein structure models

Declared that transmembrane member 16A (TMEM16A), a component of hepatocyte Ca 2+ -activated chloride channel, exacerbates hepatic I/R injury through the promotion of GPX4-dependent ferroptosis, and interrupting the TMEM16A-GPX4 interaction or inhibiting TMEM16A in liver cells may represent promising therapeutic strategies for the treatment of hepatic I/R injury [416]

ReceptorLigand Binding Assays The peptide serves as a selective ligand in receptor binding studies to evaluate GHSR-1a affinity, receptor density, and activation thresholds under controlled assay conditions

As ES cells are pluripotent and nonmalignant, they provide an ideal system to elucidate core mechanisms underlying cellular adaptation to AA stress, independent of oncogenic transformation