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Plasma lipoproteins can be separated on the basis of their electrophoretic mobility, as shown in Figure 18.15, or on the basis of their density by ultracentrifugation

Foxp3 can upregulate the activity of complexes in the electron transport chain, reprogram and regulate Treg metabolism, drive Tregs to adopt the FAO-OXPHOS metabolism model, and enhance resistance to lipotoxicity (Howie et al., 2017a, 2019)

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The interaction between the hydrolases and the CW is crucial for their activity, and their subcellular localization regulated at the post-translational level reflects their targeting mechanisms (Vollmer et al., 2008b)
