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Description
Host selenoproteins may be targeted at the protein level by the SARS-CoV-2 main protease (Mpro) Another mechanism by which SARS-CoV-2 could interfere with host selenoprotein function is by targeting it at a post-translational stage, via proteolytic degradation
Multiplexing these systems allows measuring oxidative stress and cytotoxicity in the same well with minimal alterations to the protocols
The mechanism of excessive intestinal inflammation in necrotizing enterocolitis: an immature innate immune response
Effective therapy must not wait for detailed diagnostic tests
