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It should be emphasized that a review of clinical trials over the past 5 years shows that more and more attention is being paid to non-amyloid targets, including possible treatments of inflammation, protection of synapses and neurons, regulation of vascular factors, mitochondrial function and oxidative stress, lipid and glucose metabolism, circadian rhythm, and neurogenesis, as well as epigenetic interventions [40]

The inflammatory feed-forward loop triggered by the complement component C3 as a potential target in endometriosis

More importantly, several studies demonstrated that stress hormones could promote tumour recurrence by influencing CSC functionality [249] and their reactivation from dormancy [250]