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Description
To do this, we first examined the impact of depletion of reactive oxygen species (ROS), via systemic deletion of the main source of ROS in phagocytes, NADPH oxidase (Nox)2, which we found to suppress prostate tumor growth in bone
We then investigated the activities of key enzymes in the phenylpropanoid biosynthesis pathway, hormone levels, and the expression levels of related genes at different time points
NAD+ plays a vital role in redox reactions and sirtuin activation
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